Copilot for Biomedical Writing

Ask, verify, and refine evidence in real time.


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Ask • Verify • Refine
Rapid Evidence Workflow

Ask

From idea to living review

1. Enter your topic.
2. Receive a PRISMA-grade review.
3. Get evidence-based answers to any question on your topic.

Verify

Instant fact-check of every statement

1. Paste your claim.
2. Spot unverified or contradicting data.
3. Review sources in one click.

Refine

Accept fixes, drop in citations

1. Confirm verified claims.
2. Insert citations automatically.
3. Update the document instantly.


Self-Updating Review

Synthory AI performs fully automated processing of biomedical scientific papers.


Our AI platform generates end-to-end systematic reviews with ~90% alignment to peer-reviewed publications.

Layer 1100%86%SPeer-reviewedpublicationSynthory AI86%24%17%GPT-o1Deep ResearchGeminiDeep ResearchComparison report

Examples of Generated Reviews

Our AI-generated research review shows a close match to a peer-reviewed article. To put our AI to the test, we replicated a study from a peer-reviewed journal to see how our results compared to traditional research.

Despite using different methods (manual vs. AI-driven), the results were remarkably similar, showing that AI-driven reviews can effectively mirror traditional systematic reviews. Read the report for more details.

See more our reviews published on Zenodo repository.


Contact us with inquiries or ideas




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Pilot Program

 
1. Participants will receive a biomedical systematic review.
2. We may showcase the generated reviews unless participants request full confidentiality.
3. Participation is subject to approval by Synthory AI.


© Synthory AI. All rights reserved.

Thank you for your participation in our pilot program!
Once your application is approved, we'll be in touch shortly.
Get ready to accelerate your research with comprehensive reviews!

This report provides researchers and medical-affairs teams with actionable insights into antioxidant-based therapeutic strategies for neurodegenerative diseases. Three illustrative findings are highlighted below, drawn from a comprehensive 494-page analysis; additional insights are provided in the full report.

  • TG15‑132, a selective Nox2 inhibitor, reduces ROS and inflammatory gene expression while penetrating the brain effectively—making it a strong candidate for oxidative stress–linked neurodegeneration.

  • ApoE4 expression impairs neuronal mitochondria, reducing ATP and elevating ROS, suggesting the need for mitochondrial‑targeted interventions to counteract ApoE4‑related cognitive decline.

  • Antioxidant therapy in MS patients cut ROS by 15% and reduced lipid peroxidation and DNA oxidation by 41% and 32%, respectively—supporting clinical use to manage oxidative injury and inflammation.

TitleAuthorYearISBN
6603
Records screened
784
Full-text studies included
Jan 2018–Mar 2025
Coverage window
 

Targeting Oxidative Stress in Neurodegenerative Diseases: Report
$19.00

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